
- English
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A Perspective on the Neurobiological Basis of Autism
About this book
This unique book explores the validity of early neuroinflammation as the cause of autism.
If in his previous book, the author had focused on the evolution of the diagnosis of autism over the last century, delving into the psychological, epidemiological and sociological aspects, in this book - the most comprehensive and articulate on this aspect - he has concentrated on the synthesis of research by deepening the discussion of topics related to genetic research, heavy metals, the process of infection and neuroinflammation, and the BDNF thesis. It covers the most accepted scientific theories, providing an overview of new frontiers of research. Finally, it introduces new strategies and tools for diagnosis and new frontiers of intervention.
It will be a useful resource for professionals and researchers in autism spectrum disorders and neurobiology. It will also be an interesting read for undergraduate and postgraduate students of autism, neuroscience and genetics.
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Information
Table of contents
- Cover
- Half-Title Page
- Title Page
- Copyright Page
- Table of Contents
- List of Abbreviations
- 1 From Autism Research between Psychology and Neuroscience to A Perspective on the Neurobiological Basis of Autism
- 2 The Role of BDNF in the Onset of Autism
- 3 The Origin of Connectopathy: A Circuit Hypothesis of Neuroinflammation and BDNF Dysregulation in the Onset of Autism
- 4 Genetics, Epigenetics and Environment in Autism
- 5 Genetic Research on Autism: The ‘Genetic Susceptibility Basis’
- 6 The Central Role of Shank Family Genes in Inflammation and Autism
- 7 Cerebral Characteristics in Autism and the Role of Shank Family Genes
- 8 Shank Genes, Zinc and Glutamate Receptors in the Aetiology of Autism
- 9 Dopamine Receptors in the Aetiology of Autism
- 10 The Role of Zinc and the Immune System in the Aetiology of Autism
- 11 Inflammatory Processes in the Pathogenesis of Autism
- 12 The Role of Zinc in Inflammation Process in the Aetiology of Autism
- 13 Prenatal Infection and Inflammation in the Aetiology of Autism
- 14 The Role of Metals and Trace Elements in the Aetiology of Autism
- 15 Pathomechanism of Metals in Autism
- 16 The Role of Susceptibility-Inducing Cofactors in the Aetiology of Autism: Acetaminophen, Paracetamol and Glyphosate
- 17 New Diagnostic Tools (Not Only) for Autism
- 18 New Therapeutic Intervention Tools (Not Only) for Autism
- 19 Conclusions
- Names Index
- Index