- 174 pages
- English
- ePUB (mobile friendly)
- Available on iOS & Android
eBook - ePub
About this book
Finding the time to revise effectively is one of the greatest challenges that modern MRCP candidates face. This unique book by a recent successful candidate provides precisely what many have needed but been unable to find until now: the distilled essential knowledge required to pass the MRCP Part 2 exam. This book provides candidates with a concise and well-directed approach to fast and efficient revision, supplying comprehensive coverage of the MRCP syllabus and the key points of each topic. It stands apart from other MRCP revision resources in supplying both concise text and the essential lists needed to pass the Part 2 exam.
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Yes, you can access MRCP Part 2 Examination by Ashley Bond in PDF and/or ePUB format, as well as other popular books in Medicine & Medical Theory, Practice & Reference. We have over one million books available in our catalogue for you to explore.
Information
Chapter 1
Cardiology
Cardiac arrhythmia
WPW
Type A:
- left sided accessory fibres
- right axis deviation
- dominant R wave in V1.
Type B:
- right sided accessory fibres
- left axis deviation
- non-dominant R wave in V1.
Associations:
- HOCM
- mitral valve prolapse
- Ebstein’s anomaly
- secundum ASD.
Treatment – Flecanide or Amiodarone, can be used in acute phase. Ultimately ablation is gold standard. Avoid treatment with Verapamil or Digoxin, they will increase conduction down the accessory pathway and may accelerate arrhythmias.
AF and anticoagulation1
Paroxysmal – terminate spontaneously, lasts < 7 days.
Persistent – not self-terminating, lasts > 7 days, responds to chemical or DC cardioversion.
Permanent – continuous, does not respond to cardioversion.
CHADS2:
- C – CCF/Coronary disease = 1 point
- H – Hypertension = 1 point
- A – Age > 75 years = 1 point
- D – Diabetes = 1 point
- S2 – Stroke or TIA = 2 points.
0 points = aspirin.
1 point = aspirin (could consider Warfarin).
2 points = Warfarin, with an INR range of 2–3.
AF: rate versus rhythm control
Factors favouring rate control – > 65 years old:
- history of IHD.
Factors favouring rhythm control – < 65 years old:
- symptomatic
- first presentation
- correctable precipitant.
Long QT syndrome
- Due to delayed repolarisation of the ventricles.
- QT interval > 450 ms corrected in females, > 470 ms corrected in males.
- Caused by defect in alpha-subunit of potassium channel, resulting in their blockade.
Causes:
- congenital – uses genetic classification LQT1-10, examples of which are:
- – Jervell-Lange-Nielsen (recessive, associated with deafness)
- – Romano-Ward (dominant, chromosome 11).
- drugs – Amiodarone, Sotalol, TCA, Chloroquine, Erythromycin, Antipsychotics, Antihistamine (if given with cP450 inhibitors)
- electrolytes – hypokalaemia, hypocalcaemia, hypomagnesaemia
- others – SAH, hypothermia, MI, myocarditis.
Treatment:
- acquired – remove precipitant give IV potassium and IV Mg
- for congenital use Beta Blockers and ICD for high risk patients
- Digoxin causes shortening of the QT interval, but not used in the treatment of LQTS.
Indications for temporary pacing
- Symptomatic/haemodynamically unstable bradycardia.
- Second degree or CHB following MI.
- Trifascicular block prior to surgery or if symptomatic.
Inferior MI may give rise to heart block as the Posterior interventricular supplies the AV node. This is not a definite indication for temporary pacing.
Bradycardia has an increased risk of asystole if:
- HR < 40 bpm
- SBP < 100 mmHg
- LV dysfunction
- ventricu...
Table of contents
- Cover Page
- Half Title
- Title Page
- Copyright Page
- Contents
- Preface
- List of contributors
- 1 Cardiology
- 2 Respiratory
- 3 Gastroenterology
- 4 Infectious disease and GUM
- 5 Pharmacology and toxicology
- 6 Ophthalmology
- 7 Renal
- 8 Haematology and oncology
- 9 Neurology
- 10 Rheumatology
- 11 Endocrine and diabetes
- References
- Bibliography