Vitamin E in Health and Disease
eBook - PDF

Vitamin E in Health and Disease

  1. 110 pages
  2. English
  3. PDF
  4. Available on iOS & Android
eBook - PDF

Vitamin E in Health and Disease

About this book

In this book, Vitamin E in Health and Disease, the chapter by Dr Lisa Schmölz et al., The Hepatic Fate of Vitamin E, includes the hepatic metabolism of vitamin E, its storage, release, distribution, and its effects on the metabolism in great detail, as well as its effect on the prevention of diseases, in addition to its role in anti-aging. The chapter by Dr Rusu Anca Elena reports on the effect of vitamin E in patients with hemodialysis. In a similar manner, the chapter of Drs Rayan Ahmed and Paul W. Sylvester describe g-Tocotrienol, a natural isoform within the vitamin E family of compounds, which displays potent antiproliferative, apoptotic and reversal of epithelial-to-mesenchymal-transition activity against breast cancer, employing treatment doses that have little or no effect on normal cell viability. The chapter by Milka Mileva and Angel S. Galabov describes how vitamin E could be recommended as a reliable agent, indeed as a component in multiorgan flu therapy. Last, Dr Juan José Godina-Nava et al. describe the cytoprotector effect of the 120-Hz electromagnetic fields in early hepatocarcinogenesis.

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Yes, you can access Vitamin E in Health and Disease by Jose Antonio Morales-Gonzalez in PDF and/or ePUB format, as well as other popular books in Biological Sciences & Biochemistry. We have over one million books available in our catalogue for you to explore.

Information

Table of contents

  1. Vitamin E in Health and Disease
  2. Contents
  3. Preface
  4. Chapter 1 The Hepatic Fate of Vitamin E
  5. Chapter 2 Vitamin E in Hemodialysis Patients
  6. Chapter 3 Cytoprotective Effect of 120 Hz Electromagnetic Fields on Early Hepatocarcinogenesis: Experimental and Theoretical Findings
  7. Chapter 4 Vitamin E and Influenza Virus Infection
  8. Chapter 5 - γ-Tocotrienol Reversal of Epithelial-to-Mesenchymal Transition in Human Breast Cancer Cells is Mediated through a Suppression of Canonical Wnt and Hedgehog Signaling