
- 328 pages
- English
- ePUB (mobile friendly)
- Available on iOS & Android
eBook - ePub
New Concepts in Cerebral Ischemia
About this book
Approximately half a million Americans suffer brain ischemic episodes every year. These brain strokes are the third leading cause of death in the United States and the number-one cause of permanent disability. Over the past decade, we have made extensive attempts to understand the biological mechanisms that underlie stroke-induced neuronal injury. Providing a detailed description of the techniques and theories employed in both basic and clinical research, New Concepts in Cerebral Ischemia delineates the mechanisms underlying neuronal damage induced by ischemia. The book covers methods to elucidate the pathogenesis of ischemia, technologies used in the design of specific strategies to protect neurons from further injury, and new approaches to treat patients with acute ischemic episodes. It includes methods and therapeutic approaches for neuronal protection against acute and chronic ischemic episodes and describes new animal models for ischemia research including the application of transgenic animals.
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Please note we cannot support devices running on iOS 13 and Android 7 or earlier. Learn more about using the app.
Yes, you can access New Concepts in Cerebral Ischemia by Rick C. S. Lin in PDF and/or ePUB format, as well as other popular books in Medicine & Biology. We have over one million books available in our catalogue for you to explore.
Information
Table of contents
- Cover
- Half Title
- Title Page
- Copyright Page
- Methods & New Frontiers in Neuroscience
- Preface
- The Editor
- Contributors
- Table of Contents
- 1 Gender Differences in Acute Cerebral Ischemia: Neuroprotective Mechanisms of Estrogen
- 2 Neonatal Hypoxic-Ischemic Brain Insults and Their Mechanisms
- References
- 3 Neural Stem Cell Biology Provides Insights into New Therapeutic Strategies for Hypoxic-Ischemic Brain Injury
- 4 Excitotoxicity, Oxidative Stress, and Apoptosis in Ischemic Neuronal Death
- 5 Intracellular Ca2+ Signals Underlying Rapid and Delayed Excitotoxicity I Mature CNS
- 6 Mechanism of Zinc-Induced Neuronal Death
- 7 Cerebral Ischemia and Adenosine: “Spheres of Action”*
- 8 Calpain and Caspase in Ischemic and Traumatic Brain Injury
- 9 Brain Inflammation, Cytokines, and p38 MAP Kinase Signaling in Stroke
- 10 The Potential Role of Hyperbaric Oxygen in the Treatment of Stroke
- 11 Cellular and Molecular Mechanisms of Ischemic Tolerance
- Index