Edhf 2000
eBook - ePub

Edhf 2000

  1. 544 pages
  2. English
  3. ePUB (mobile friendly)
  4. Available on iOS & Android
eBook - ePub

Edhf 2000

About this book

Understanding the nature and role of endothelium-derived hyperpolarizing factor appears to be crucial in the quest for improved treatments for hypertension, diabetes, ischemia-reperfusion and other vascular disorders.
EDHF 2000 comprises the proceedings of the Third International Symposium on endothelium-dependent hyperpolarizations. The first t

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Yes, you can access Edhf 2000 by Paul M. Vanhoutte in PDF and/or ePUB format, as well as other popular books in Medicine & Anatomy. We have over one million books available in our catalogue for you to explore.

Information

Publisher
CRC Press
Year
2003
eBook ISBN
9781040161968
Edition
0
Topic
Medicine
Subtopic
Anatomy

Table of contents

  1. Cover
  2. Half Title
  3. Title Page
  4. Copyright Page
  5. Table of Contents
  6. Preface
  7. Acknowledgements
  8. Contributors
  9. 1 What do gap junctions do anyway?
  10. 2 Cardiovascular gap junctions: functional diversity, complementation and specialization of connexins
  11. 3 Endothelium and smooth muscle pathways for conduction along resistance microvessels
  12. 4 Membrane potential and calcium responses evoked by acetylcholine in submucosal arterioles of the guinea-pig small intestine
  13. 5 The effects of ouabain, 18a-glycyrrhetinic acid and connexin-mimetic peptides on intercellular communication in cells expressing a Cx43-GFP chimeric protein
  14. 6 Role of gap junctions in endotheliumdependent hyperpolarizations
  15. 7 Heterogeneity of EDHF-type relaxations of rabbit and rat arteries analysed with peptides homologous to the extracellular loops of connexins 37, 40 and 43
  16. 8 Myoendothelial and circumferential spread of endothelium-dependent hyperpolarization in coronary arteries
  17. 9 Direct myoendothelial contacts in human pulmonary microvessels
  18. 10 Role of gap junctional communication in EDHF-mediated responses and mechanisms of K+-induced dilatations
  19. 11 Comparison of a and ß isoforms of glycyrrhetinic acid and carbenoxolone as inhibitors of EDHF-type relaxation
  20. 12 Inhibitory effect of 18-/3-glycyrrhetinic acid on the relaxation induced by acetylcholine in the rat aorta
  21. 13 A central role for endothelial cell potassium channels in EDHF-mediated responses
  22. 14 Could the EDHF be K+ in porcine coronary arteries?
  23. 15 Nitro-L-arginine/indomethacin-resistant relaxations to acetylcholine in small gastric arteries of the rat: Effect of ouabain plus Ba2+ and relation to potassium ions
  24. 16 Effects of barium, ouabain and K+ on the resting membrane potential and endothelium-dependent responses in rat arteries
  25. 17 EDHF and potassium: blockade of chloride channels reveals relaxations of rat mesenteric artery to potassium
  26. 18 Cytochrome P450 2C - a source of EDHF and reactive oxygen species in the porcine coronary artery
  27. 19 Cortisol increases EDHF-mediated relaxations in porcine coronary arteries and up-regulates the expression of cytochrome P450 2C9
  28. 20 An arachidonic acid metabolite(s) produced by the endothelial cytochrome P450 isoform, CYP3A4, relaxes the lingual artery of the monkey via K+ channel opening
  29. 21 EDHF-mediated responses induced by bradykinin in the porcine coronary artery
  30. 22 Epoxyeicosatrienoic acid release mediates nitric oxide-independent dilatation of rat mesenteric vessels
  31. 23 Epoxyeicosatrienoic acid and endothelium-dependent hyperpolarization in porcine coronary arteries
  32. 24 Lipoxygenase-derived metabolites of arachidonic acid are not involved in the endothelium-dependent hyperpolarization to acetylcholine in the carotid artery of the guinea-pig
  33. 25 Cytochrome P450 isoforms in the brain encode cell specific hyperpolarizing factors with a common mechanism of action
  34. 26 Expression of recombinant cytochrome P450 epoxygenase in rat brain
  35. 27 Identification of 11,12,15-trihydroxyeicosatrienoic acid as the mediator of acetylcholine-and arachidonic acid-induced relaxations in the rabbit aorta
  36. 28 Identification of hydrogen peroxide as an endothelium-derived hyperpolarizing factor in mice
  37. 29 Components of the potassium currents underlying the actions of endothelium-derived hyperpolarizing factor in arterioles
  38. 30 Evidence for relaxation to endothelium-derived hyperpolarizing factor (EDHF) in isolated small mesenteric arteries of the mouse
  39. 31 Pharmacological characterization of potassium channels in intact mesenteric arteries and single smooth muscle cells from eNOS-I- and +/+ mice
  40. 32 EDHF, which is not NO, is a major endothelium-dependent vasodilator in mice
  41. 33 Prostacyclin and iloprost in the isolated carotid artery of the guinea-pig
  42. 34 Role of charybdotoxin/apamin sensitive Kja channels in pulsatile perfusion-mediated coronary vasodilatation in vivo
  43. 35 Essential role of estrogen in the EDHF-mediated responses of mesenteric arteries from middle-aged female rats: possible contribution of gap junctional protein connexin43
  44. 36 Endothelium-derived hyperpolarizing factor (EDHF) and utero-feto-placental circulation in the rat
  45. 37 Endothelium-derived hyperpolarizing factor maintains a normal relaxation to bradykinin despite impairment of the nitric oxide pathway in porcine coronary arteries with regenerated endothelium
  46. 38 Mechanisms underlying the vasodilatation caused by bradykinin in essential hypertensive patients
  47. 39 Influence of diabetes on endotheliumdependent responses in mesenteric and femoral arteries of rats
  48. 40 Folate restores the NO synthase-and cyclooxygenase-resistant renal vasodilator response to acetylcholine in diabetes
  49. 41 Resistance of EDHF-mediated relaxations to oxidative stress in human radial arteries
  50. 42 Critical limb ischemia results in different types of endothelial dysfunction depending on the vascular bed studied
  51. 43 Potentiated EDHF-mediated dilatations in the rat middle cerebral artery following ischemia/reperfusion
  52. 44 The EDHF-dependent but not the NO-dependent component of the acetylcholine-induced relaxation of the rabbit aorta is resistant to ionized radiation
  53. 45 Inhibition of converting enzyme prevents the age-related decline in endothelium-dependent hyperpolarization
  54. 46 Effects of a converting enzyme inhibitor, an ATj-receptor antagonist and their combination on endothelial dysfunction in hypertension
  55. 47 EDHF: gap junction or chemical? and many other questions
  56. References
  57. Index